I agree with Carl. I don’t avoid fluoride 100%, only in my drinking water and medications. Coincidentally, I’ve had serious side effects from fluoride-containing medications (Levofloxacin, Fluoxetine).
In 2015, six psychiatrists from the Department of Psychiatry, University of Oxford, the UK, published the study, “Depression and Violence: A Swedish Population Study” in Lancet Psychiatry.
“. . . if the major media picks up on this story, they will have the chance to report on what arguably is the worst—and most harmful—scandal in American medical history”
Historically, there have always been some patients who report that any treatment for depression—including bloodletting—has worked for them, but science demands that for a treatment to be deemed truly effective, it must work better than a placebo or the passage of time without any treatment. This is especially important for antidepressant drugs—including Prozac, Zoloft, and other selective serotonin reuptake inhibitors (SSRIs), as well as Effexor, Cymbalta, and other serotonin and norepinephrine reuptake inhibitors (SNRIs)—because all of these drugs have uncontroversial troubling side effects.
Yet one glaring problem remains. Despite promising clinical results, no one knows exactly how psychedelic drugs work in the brain. Examining their actions on brain cells isn’t just an academic curiosity. It could give rise to variants that maintain antidepressant properties without the high. And because hallucinogens substantially alter our perception [management?!] of the world, they could be powerful toolsfor investigating the neurobiology behind consciousness.
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This year, scientists found another common theme—psychedelics seem to “reset” the brain to a more youthful state, at least in mice. Like humans, mice have an adolescent critical period, during which their brains are highly malleable and can easily rewire neural circuits, but the window closes after adulthood.
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An earlier study showed that MDMA reopens the critical window in adult mice, so that they change their “personality.” Mice raised alone are often introverted and prefer to keep to themselves in adulthood. A dose of MDMA increased their willingness to snuggle with other mice—essentially, they learned to associate socializing with happiness, concluded the study.
It’s not that surprising. MDMA is well-known to promote empathy and bonding. The new study, by the same team, extended their early results to four psychedelics that don’t trigger fuzzy feelings—LSD, ketamine, psilocybin, and ibogaine. Similar to MDMA, adult mice raised alone changed their usual preference for solitude when treated with any of the drugs. Because habits are hard to change in adulthood—for mice and men—the drugs may have reopened the critical period, allowing the brain to more easily rewire neural connections based on new experiences.
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These are just early results. But psychedelic research is gaining a new ally—artificial intelligence. Algorithms that predict protein structure, combined with rational drug design, could generate psychedelics that retain their psychiatric benefits without the high.
A recent study published in the journal Molecular Psychiatry sent shockwaves across the scientific community and popular outlets as it disproved the predominant “serotonin hypothesis” of depression. In just two weeks since its publication it has been accessed by nearly half a million people and the subject of dozens of subsequent articles. The researchers analyzed a total of seventeen systematic reviews, meta-analyses, and other large studies focused on the following six tenets pertinent to the “serotonin hypothesis” of depression:
Ms. Cat’s Chronicles 
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